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In experiments of other receptor tyrosine kinases implicated from the oncogenesis of GIST, nilotinib realized strong and selective inhibition of PDGFR? and PDGFR?. As is the case with imatinib, nilotinib potently inhibited the autophosphorylation of A31 cells remodeled by PDGFRALower amounts of potassium or magnesium while in the blood can also inc

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